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Glucose trafficking through coupled astrocytes is necessary for hyperactivity, while extracellular glucose deprivation causes loss of synaptic hyperactivity that can be rescued when astrocytes are filled with glucose or lactate, showing the importance of metabolism in epilepsy formation

Next, there is partial disassembly of the PSII core complex leading to the release of CP43 inner antenna protein, OEC proteins, and other unknown low molecular mass subunits followed by proteolytic degradation from D1

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The broad application of NAC is not only because of its well-described antioxidant and radical scavenging activity but also because, as a thiol molecule, it is quite stable, commercially available, and inexpensive